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【单选题】
依据《建设项目竣工环境保护验收技术规范——生态影响类》,以下不属于需特殊保护地区的是(    )。
A.
饮用水水源保护区、自然保护区、风景名胜区、生态功能保护区、基本农田保护区、水土流失重点防治区、森林公园、地质公园、世界遗产地、国家重点文物保护单位、历史文化保护地等
B.
国家法律、法规、行政规章及规划确定的或经县级以上人民政府批准的需要特殊保护的地区
C.
有特殊价值的生物物种资源分布区域
D.
沙尘暴源区
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【单选题】It is predicted by experts that the number of animals used for toxicity testing in Europe will quintuple due to a plan to have a large variety of chemical tested.
A.
Y
B.
N
C.
NG
【单选题】toxicity
A.
修改
B.
治疗
C.
毒效
【判断题】2,4-D施用后两天内不要修剪草坪。
A.
正确
B.
错误
【单选题】若信号量S的初值为3,当前值为-1,则表示有( )等待进程。
A.
0个
B.
1个
C.
2个
D.
3个
【单选题】若信号量S的初值为3,当前值为-2,则表示有( )等待进程。
A.
2个
B.
3个
C.
4个
D.
5个
【多选题】What is lipid toxicity hypothesis?
A.
According to the “lipid toxicity” hypothesis, the action of PPARγ on adipocytes normally keeps the cells ready to synthesize and store triacylglycerols—the adipocytes are insulin-sensitive and produce leptin, which leads to their continued intracellular deposition of TAGs.
B.
However, excess caloric intake in obese individuals causes adipocytes to become filled with TAGs, leaving adipose tissue unable to meet any further demand for TAG storage. Lipid-filled adipose tissue releases protein factors that attract macrophages, which infiltrate the tissue and may eventually represent as much as 50% of the adipose tissue by mass.
C.
Macrophages trigger the inflammatory response, which impairs TAG deposition in adipocytes and favors release of free fatty acids into the blood. These excess fatty acids enter liver and muscle cells, where they are converted to TAGs that accumulate as lipid droplets. This ectopic deposition of TAGs leads to insulin insensitivity in liver and muscle, the hallmark of type 2 diabetes.
D.
According to this hypothesis, excess stored fatty acids and TAGs are toxic to liver and muscle. Some individuals are less well equipped genetically to handle this burden of ectopic lipids and are more susceptible to the cellular damage that leads to development of type 2 diabetes. Insulin resistance probably involves impairment of several of the mechanisms by which insulin acts on metabolism, which include changes in protein levels and changes in the activities of signaling enzymes and transcription factors. For example, both adiponectin synthesis in adipocytes and adiponectin levels in the blood decrease with obesity and increase with weight loss.
【简答题】Chronic toxicity
【简答题】toxicity
【单选题】若信号量的初值为2,当前值为-3,则表示有( )个进程在等待。
A.
1
B.
2
C.
3
D.
4
【简答题】Oral toxicity
相关题目:
【多选题】What is lipid toxicity hypothesis?
A.
According to the “lipid toxicity” hypothesis, the action of PPARγ on adipocytes normally keeps the cells ready to synthesize and store triacylglycerols—the adipocytes are insulin-sensitive and produce leptin, which leads to their continued intracellular deposition of TAGs.
B.
However, excess caloric intake in obese individuals causes adipocytes to become filled with TAGs, leaving adipose tissue unable to meet any further demand for TAG storage. Lipid-filled adipose tissue releases protein factors that attract macrophages, which infiltrate the tissue and may eventually represent as much as 50% of the adipose tissue by mass.
C.
Macrophages trigger the inflammatory response, which impairs TAG deposition in adipocytes and favors release of free fatty acids into the blood. These excess fatty acids enter liver and muscle cells, where they are converted to TAGs that accumulate as lipid droplets. This ectopic deposition of TAGs leads to insulin insensitivity in liver and muscle, the hallmark of type 2 diabetes.
D.
According to this hypothesis, excess stored fatty acids and TAGs are toxic to liver and muscle. Some individuals are less well equipped genetically to handle this burden of ectopic lipids and are more susceptible to the cellular damage that leads to development of type 2 diabetes. Insulin resistance probably involves impairment of several of the mechanisms by which insulin acts on metabolism, which include changes in protein levels and changes in the activities of signaling enzymes and transcription factors. For example, both adiponectin synthesis in adipocytes and adiponectin levels in the blood decrease with obesity and increase with weight loss.